Comparison of the responses to drugs acting on adrenoreceptors and muscarinic receptors in human isolated corpus cavernosum and cavernous artery. Contemporary Management of Impotence and Infertility. Neuroanatomy and neurophysiology of penile erection. Philadelphia, Pa: Elsevier Saunders 2012.ĭe Groat WC, Steers WD. Wein AJ, Kavoussi LR, Novick AC, Partin AW, Peters CP, eds.
COMPLETE ANATOMY KEEP SURROUNDING STRUCTURES FULL
The tunica albuginea is stretched to its capacity, compressing emissary veins and thus further decreasing venous outflow as a result, intracavernous pressure increases and is further increased by contraction of the ischiocavernous and bulbospongiosus muscles, resulting in full rigidity Subtunical venular plexuses are compressed between the tunica albuginea and the distended sinusoids, leading to decreased venous outflow The sinusoids within the corpora cavernosa distend with blood With relaxation of the smooth muscles in the trabeculae and the arterial wall, the following events occur, which lead to an erection:Īrterial inflow increases as a result of dilatation of the arterioles and arteries Other neurotransmitters, including vasoactive intestinal peptide (VIP), calcitonin gene-related peptide (CGRP), prostaglandins, and other peptides, may also be involved in the erectile process. The release of NO increases the production of cyclic guanosine monophosphate (cGMP), which relaxes cavernosal smooth muscle. Nonadrenergic, noncholinergic (NANC) neurons release NO. Nitric oxide (NO) appears to be the principal neurotransmitter causing penile erection.
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